![]() ![]() An unenhanced abdominal and pelvic CT showed mild pancreatic tail enlargement, increased attenuation of the peripancreatic fat, and a pancreatic-peripancreatic collection contacting the posterior gastric wall ( Figure 1A and B). Gut 2004 53 1340-1344., hematocrit 43.8%, hemoglobin 15.5 gr/dl, C-reactive protein 2.3 mg/dl, creatinine 2.18 mg/dl, amylase 168 U/l, lipase 53 U/l, and normal liver tests, plasma electrolytes and coagulation tests. Persistent organ failure during the first week as a marker of fatal outcome in acute pancreatitis. ![]() The laboratory examination results were: leukocytes: 6,080 cells/mm 3 3 Johnson C, Abu-Hilal M. He was with heart rate of 91 bpm, blood pressure of 150/69 mmHg, temperature of 37.3° C, pale skin and mucous membranes, soft abdomen sensitive at the epigastrium. CASE REPORTĤ3-year-old male with intense epigastric abdominal pain with an episode of hematemesis looked for medical assistence. ![]() The aims of the present paper were: 1) report a clinical case with a rare presentation of AP (hematemesis) with a slow evolution towards gastric perforation that was remarkable for the absence of celiac axis thrombosis as evidenced by imaging and 2) review AP in terms of clinical presentation, imaging, risk factors, complications and treatment. Gastric perforation is a rare complication of AP. Acute pancreatitis(AP) have a high morbidity and mortality. ![]()
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